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ECG.CASES

@ecgcases

Daily ECG

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Post #796 6.42K
65 Y female presented with palpitation
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Post #795 5.97K
#case183 ❤️
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Post #794 6.54K
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Post #793 6.01K
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Post #792 5.61K
#Extra02 ❤️
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Post #791 6.12K
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Post #790 5.54K
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Post #789 5.36K
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Post #788 5.19K
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Post #787 5.27K
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Post #786 5.55K
Effects of hyperkalemia on the ECG:

K > 5.5 mEq/L
Peaked T waves (usually the earliest sign of hyperkalaemia)

K > 6.5 mEq/L
P wave widens and flattens
PR segment lengthens
P waves eventually disappear

K > 7.0 mEq/L
Wide QRS complexes with bizarre QRS morphology
ST elevation
High-grade AV block
Any kind of conduction block (bundle branch blocks, fascicular blocks)
Sinus bradycardia or slow AF
Development of a sine wave appearance

K > 9.0 mEq/L
Asystole
Ventricular fibrillation
PEA with bizarre, wide complex rhythm
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Post #785 5.28K
The correct answer is “Hyperkalemia”
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Post #784 5.55K
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Post #783 5.49K
70 y male KCO:DM, HTM and CKD presented with nausea, vomiting and confusion. the patient was hypotensive with O2 saturation of 77%
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Post #782 5.11K
#case182 ❤️
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Post #780 5.84K
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Post #779 5.5K
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Post #778 5.5K
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Post #777 5.72K
Narrow complexes at a rate of ~70 which are not preceded by P waves suggest (accelerated) #junctional_rhythm.

There are p waves right after QRS complexes with a rate fairly (not exactly) similar to the rate of QRS complexes that indicates #Isorhythmic_AV_dissociation.

There are STT changes compatible with #inferoposterior wall MI.
Inferior MI is suggested by presence of these changes in the inferior leads:
• ST-elevation
• Q wave

Posterior MI is suggested by the following changes in V1-3:
• Horizontal ST depression
• Tall, broad R waves
• Dominant R wave (R/S ratio>1)

Thanks to my friend Salvatore Pezzella for such a beautiful scheme
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Post #776 5.48K
The correct answer is “Junctional rhythm, Isorrhythmic AV dissociation and inferoposterior MI”
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